• Clean energy
  • Appetite suppression
  • Increased focus
  • Improved performance
  • Consistent weight loss
SKU: fitmax Category:


Fat loss is an important goal for achieving the body that we want. Whether your goals are based on performance, attractiveness, or overall health, losing fat can help you run faster, look better, and feel great. While there are many purported fat burners on the market, none have been formulated by carefully combining the best ingredients that have been scientifically proven to work together synergistically – until now. That is why Performax Labs is proud to bring you FitMaxTM, the fat burner designed using cutting-edge research to help you achieve your body composition goals. With scientifically-backed synergy like the patented, proven ingredient Teacrine combined with caffeine, FitMaxTM will promote optimal fat burning while providing a clean boost of energy throughout the day.

• Teacrine – Derived from tea, this natural purine alkaloid promotes cognitive enhancement, providing clean, all-day energy.
• Caffeine – A popular stimulant with fat burning effects, it also increases the concentration and effectiveness of Teacrine when combined.
• Carnitine – Carnitine is an essential transporter of fatty acids into mitochondria where it can be metabolized and used for energy.
• Choline – An essential brain nutrient, choline can improve saturation of carnitine in muscle tissue to optimize its fat burning potential.
• Citrus Aurantium – Bitter orange is a natural source of synephrine, which has been shown to enhance weight loss via increased calorie burn.
• Rhodiola rosea – A popular adaptogen, Rhodiola has demonstrated synergistic benefits with Citrus aurantium, resulting in enhanced fat loss and neurotransmitter action.

The keys to this formula are in its precise combinations of ingredients backed by science. For example, Rhodiola rosea provides anti-stress and adaptogenic benefits that pair perfectly with the neurotransmitter enhancement of Citrus aurantium. Therefore, the effects of both are enhanced so that more fat is burned, and the increased clarity and focus feel cleaner with no side effects. Overall, the result is optimized body composition with a focus and energy boost that is clear and long-lasting. If fat loss is your goal, then choose the science and synergy of FitMaxTM.

Additional information

Weight 291 g
Dimensions 9 x 9 x 9 cm

Pink Lemonade, Orange Mango

Supplement Facts


Q: How do I take Performax Labs’ FitMax?
A: Take one serving (one scoop) daily in the morning. For a super boost of energy, take two scoops. However, everyone react differently, so always start with one to ascertain your body’s level of tolerance.

Q: What other Performax Labs’ products can I stack with FitMax?
A: Since FitMax provides clean, long-lasting energy, it stacks really well with VasoMax as a pre and IntraMax as your during workout beverage.

Q: How can synergy be scientifically proven?
A: For proven synergy, compounds must be studied together and alone, so that comparisons can be made. The compounds may enhance each other’s action or concentration in the body, or both.

Q: When is the best time to take Performax Labs’ FitMax?
A: It is best to take FitMax in the morning to set metabolism for the day and keep sleep optimal. FitMax can be taken as a morning coffee replacement or as a pre-workout, but should be avoided in the evening hours.


L-Carnitine L-Tartrate
Carnitine is essential for the body to transport fatty acids into the mitochondria to be metabolized. Not only will the mitochondria function better overall with supplemental carnitine (which equals better metabolism), but more fat will be transported and burned.

• Malaquarnera et al. (2007) showed that supplementation with carnitine can reduce fat mass while increasing muscle mass in subjects.

Choline Bitartrate
Choline is an important brain nutrient that serves as a precursor to acetylcholine. Supplemental choline improves weight loss, and it works synergistically with carnitine to keep carnitine levels high in muscle tissue for metabolic optimization.

• After one week of choline supplementation, weight-class athletes (martial artists) were able to rapidly lose weight in a safe manner while strength remained intact (Elsawy et al. 2014).
• Daily et al. (1995) determined that combining carnitine with choline supports tissue maintenance of carnitine and reduced excretion to enhance fat mobilization and optimal metabolism.

Olive Leaf Extract (Std. to 40% Oleuropein)
Olive leaf extract has shown potent fat loss properties through a variety of mechanisms. Not only can olive leaf increase thyroid hormones, which regulate metabolism, but it also increases uncoupling proteins, which activate production of brown adipose tissue – the fat-burning fat cells!

• Shen et al (2014) showed that olive leaf extract can reduce body weight, reduce fat in the blood and can positively affect many thermogenic and adipogenic (fat-causing) genes.

Rhodiola Rosea (Std. to 3% Rosavins and 2% Salidroside)
One of the most popular adaptogenic botanicals, rhodiola is typically utilized to reduce fatigue and improve cognition. Recent evidence has shown that its anti-stress mechanisms work synergistically with other compounds to supercharge weight loss.

• Verpeut et al. (2013) demonstrated that Rhodiola rosea and Citrus aurantium have synergistic effects on belly fat (30% decrease), food intake (10.5% reduction) and neurotransmitters (15% increase in norepinephrine and 150% increase in dopamine), compared with the effects of either compound alone.

Citrus Aurantium Extract
Citrus aurantium is a natural source of p-synephrine, which is a molecule similar in structure to ephedrine. By itself or in combination with other compounds, p-synephrine has demonstrated effective weight loss in multiple studies.

• In a meta-analysis, Stohs et al. (2012) discussed the evidence validating Citrus aurantium as an effective weight loss aid, specifically regarding increased metabolic rate and energy expenditure.

Fucoxanthin (from black seaweed extract)
Fucoxathin is a carotenoid pigment found in brown and black seaweed. As a fat-soluble molecule, it is stored in fat cells for long periods of time, where it can beneficially induce fat loss and reduce fat cell differentiation and proliferation.

• Abidov et al. (2010) observed that 16 weeks of supplementation with fucoxanthin resulted in significant weight loss and increased energy expenditure (calorie burning).

Caffeine Anhydrous
Caffeine is the most widely consumed “supplement” in the world. A well-known cognitive enhancer, its effects on adrenaline, fat burning, and increasing metabolic rate make it a must in any fat-burning formula.

• Astrup et al. (1990) observed that supplementing with caffeine can increase metabolic rate by 32.4 kcal/hr, and increase fat burning.

N, N-Dimethylphenethylamine Citrate
N, N-Dimethylphenethylamine is a trace amine that modulates various neurotransmitters, similar to DMAA, with which it shares many structural similarities. Due to its specific chemical structure, N, N-Dimethylphenethylamine is safer than DMAA while still providing a powerful boost in focus and mood.

• Multiple in vitro and in vivo studies observed how phenylethylamines can increase dopamine secretion both (Sotnikova et al., 2004; Ishida et al., 2005).

Teacrine (40% Tasteless Theacrine)
Teacrine is a naturally-occurring purine alkaloid that provides an excellent boost of energy and focus with no jitters or side effects. Additionally, it has shown synergy when combined with caffeine.

• He et al. (2017) demonstrated that 150 mg of caffeine combined with 125 mg of Teacrine increases the maximum plasma concentration and area under the curve of Teacrine, with no negative effects on serum caffeine levels, resulting in synergistic benefits.

Hordeum Vulgare (std. to Hordenine)
Hordeum vulgare is a naturally-occurring source of hordenine, an alkaloid that is a trace amine active in the central nervous system. As a noradrenaline reuptake inhibitor, it increases the bioavailability and action of noradrenaline, thereby improving energy and focus.

• Frank et al. (1990) demonstrated adrenaline-like effects of hordenine in vivo, which support evidence that hordenine activates noradrenaline and its reuptake in an ex vivo model.

Huperzine-A (Huperzia Serrata 1% Extract)
Huperzine A is an acetylcholinesterase inhibitor, which means that less acetylcholine is metabolized, so high concentrations remain in the body. Acetylcholine in an important neurotransmitter for many parts of the central nervous system, but especially at the neuromuscular junction.

• Kozikowski et al. (2003) demonstrated the high affinity of huperzine A to inhibit acetylcholinesterase and that it works as well or better than commonly utilized pharmaceuticals.

Rauwolfia Vomitoria Extract (std. min. 90% rawolscine)
Rauwolfia is a natural source of rawolscine, which is a molecule very similar to yohimbine. It acts as an alpha-2 adrenergic antagonist, which induces elevated mood and energy, and weight loss.

• Perry et al. (1981) showed that rauwolscine functions as an alpha-2 adrenergic antagonist, which leads to noradrenaline release which can support weight loss and improved energy.


L-Carnitine L-Tartrate
1. Malaguarnera, M., et al., L-Carnitine treatment reduces severity of physical and mental fatigue and increases cognitive functions in centenarians: a randomized and controlled clinical trial. Am J Clin Nutr, 2007. 86(6): p. 1738-44.
2. Pistone, G., et al., Levocarnitine administration in elderly subjects with rapid muscle fatigue: effect on body composition, lipid profile and fatigue. Drugs Aging, 2003. 20(10): p. 761-7.
3. Volek, J.S., et al., L-Carnitine L-tartrate supplementation favorably affects markers of recovery from exercise stress. American Journal of Physiology-Endocrinology and Metabolism, 2002. 282(2): p. E474-E482.
4. Kraemer, W.J., et al., The effects of L-carnitine L-tartrate supplementation on hormonal responses to resistance exercise and recovery. The Journal of Strength & Conditioning Research, 2003. 17(3): p. 455-462.
5. Kraemer, W.J., et al., Androgenic responses to resistance exercise: effects of feeding and L-carnitine. Medicine & Science in Sports & Exercise, 2006. 38(7): p. 1288-1296.

Choline Bitartrate
1. Elsawy, G., O. Abdelrahman, and A. Hamza, Effect of choline supplementation on rapid weight loss and biochemical variables among female taekwondo and judo athletes. Journal of human kinetics, 2014. 40(1): p. 77-82.
2. Daily III, J.W. and D.S. Sachan, Choline supplementation alters carnitine homeostasis in humans and guinea pigs. The Journal of nutrition, 1995. 125(7): p. 1938.
3. Dodson, W.L. and D.S. Sachan, Choline supplementation reduces urinary carnitine excretion in humans. Am J Clin Nutr, 1996. 63(6): p. 904-10.
4. Hongu, N. and D.S. Sachan, Carnitine and choline supplementation with exercise alter carnitine profiles, biochemical markers of fat metabolism and serum leptin concentration in healthy women. J Nutr, 2003. 133(1): p. 84-9.
5. Naber, M., B. Hommel, and L.S. Colzato, Improved human visuomotor performance and pupil constriction after choline supplementation in a placebo-controlled double-blind study. Scientific reports, 2015. 5: p. 13188.
6. Gao, X., et al., Higher Dietary Choline and Betaine Intakes Are Associated with Better Body Composition in the Adult Population of Newfoundland, Canada. PLoS One, 2016. 11(5): p. e0155403.
7. Haubrich, D.R., et al., Increase in rat brain acetylcholine induced by choline or deanol. Life sciences, 1975. 17(6): p. 975-980.
8. Zeisel, S.H., Nutritional importance of choline for brain development. Journal of the American College of Nutrition, 2004. 23(sup6): p. 621S-626S.
9. Cohen, E.L. and R.J. Wurtman, Brain acetylcholine: control by dietary choline. Science, 1976. 191(4227): p. 561-562.
10. Zeisel, S.H., Dietary choline: biochemistry, physiology, and pharmacology. Annual review of nutrition, 1981. 1(1): p. 95-121.

Olive Leaf Extract (Std. to 40% Oleuropein)
1. Shen, Y., et al., Olive leaf extract attenuates obesity in high-fat diet-fed mice by modulating the expression of molecules involved in adipogenesis and thermogenesis. Evidence-Based Complementary and Alternative Medicine, 2014. 2014.
2. Arantes-Rodrigues, R., et al., High doses of olive leaf extract induce liver changes in mice. Food and chemical toxicology, 2011. 49(9): p. 1989-1997.
3. Poudyal, H., F. Campbell, and L. Brown, Olive leaf extract attenuates cardiac, hepatic, and metabolic changes in high carbohydrate–, high fat–fed rats. The journal of nutrition, 2010. 140(5): p. 946-953.
4. Al‐Qarawi, A., M. Al‐Damegh, and S. ElMougy, Effect of freeze dried extract of Olea europaea on the pituitary–thyroid axis in rats. Phytotherapy Research, 2002. 16(3): p. 286-287.
5. De Leonardis, A., et al., Isolation of a hydroxytyrosol-rich extract from olive leaves (Olea Europaea L.) and evaluation of its antioxidant properties and bioactivity. European Food Research and Technology, 2008. 226(4): p. 653-659.

Rhodiola Rosea (Std. to 3% Rosavins and 2% Salidroside)
1. Verpeut, J.L., A.L. Walters, and N.T. Bello, Citrus aurantium and Rhodiola rosea in combination reduce visceral white adipose tissue and increase hypothalamic norepinephrine in a rat model of diet-induced obesity. Nutrition research, 2013. 33(6): p. 503-512.
2. Gardiner, P. and M. Heuer, Nutritional composition which promotes weight loss, burns calories, increases thermogenesis, supports energy metabolism and/or suppresses appetite. 2005, Google Patents.
3. Edwards, D., A. Heufelder, and A. Zimmermann, Therapeutic Effects and Safety of Rhodiola rosea Extract WS® 1375 in Subjects with Life‐stress Symptoms–Results of an Open‐label Study. Phytotherapy Research, 2012. 26(8): p. 1220-1225.
4. Spasov, A.A., et al., A double-blind, placebo-controlled pilot study of the stimulating and adaptogenic effect of Rhodiola rosea SHR-5 extract on the fatigue of students caused by stress during an examination period with a repeated low-dose regimen. Phytomedicine, 2000. 7(2): p. 85-9.
5. Shevtsov, V.A., et al., A randomized trial of two different doses of a SHR-5 Rhodiola rosea extract versus placebo and control of capacity for mental work. Phytomedicine, 2003. 10(2-3): p. 95-105.
6. Darbinyan, V., et al., Rhodiola rosea in stress induced fatigue–a double blind cross-over study of a standardized extract SHR-5 with a repeated low-dose regimen on the mental performance of healthy physicians during night duty. Phytomedicine, 2000. 7(5): p. 365-71.

Citrus Aurantium Extract
1. Stohs, S.J., et al., Effects of p-synephrine alone and in combination with selected bioflavonoids on resting metabolism, blood pressure, heart rate and self-reported mood changes. Int J Med Sci, 2011. 8(4): p. 295-301.
2. Stohs, S.J., H.G. Preuss, and M. Shara, A Review of the Human Clinical Studies Involving Citrus aurantium (Bitter Orange) Extract and its Primary Protoalkaloid p-Synephrine. International Journal of Medical Sciences, 2012. 9(7): p. 527-538.
3. Jordan, R., et al., Beta-adrenergic activities of octopamine and synephrine stereoisomers on guinea-pig atria and trachea. J Pharm Pharmacol, 1987. 39(9): p. 752-4.
4. Arch, J.R., beta(3)-Adrenoceptor agonists: potential, pitfalls and progress. Eur J Pharmacol, 2002. 440(2-3): p. 99-107.
5. Seifert, J.G., et al., Effect of acute administration of an herbal preparation on blood pressure and heart rate in humans. Int J Med Sci, 2011. 8(3): p. 192-7.
6. Pellati, F. and S. Benvenuti, Chromatographic and electrophoretic methods for the analysis of phenethylamine [corrected] alkaloids in Citrus aurantium. J Chromatogr A, 2007. 1161(1-2): p. 71-88.
7. Servillo, L., et al., Tyramine Pathways in Citrus Plant Defense: Glycoconjugates of Tyramine and Its N-Methylated Derivatives. J Agric Food Chem, 2017. 65(4): p. 892-899.

Fucoxanthin (from black seaweed extract)
1. Kang, S.-I., et al., Petalonia binghamiae extract and its constituent fucoxanthin ameliorate high-fat diet-induced obesity by activating AMP-activated protein kinase. Journal of agricultural and food chemistry, 2012. 60(13): p. 3389-3395.
2. Abidov, M., et al., The effects of Xanthigen in the weight management of obese premenopausal women with non-alcoholic fatty liver disease and normal liver fat. Diabetes Obes Metab, 2010. 12(1): p. 72-81.
3. Hu, X., et al., Combination of fucoxanthin and conjugated linoleic acid attenuates body weight gain and improves lipid metabolism in high-fat diet-induced obese rats. Archives of biochemistry and biophysics, 2012. 519(1): p. 59-65.
4. Maeda, H., et al., Anti-obesity and anti-diabetic effects of fucoxanthin on diet-induced obesity conditions in a murine model. Molecular Medicine Reports, 2009. 2(6): p. 897-902.
5. Nishikawa, S., M. Hosokawa, and K. Miyashita, Fucoxanthin promotes translocation and induction of glucose transporter 4 in skeletal muscles of diabetic/obese KK-A y mice. Phytomedicine, 2012. 19(5): p. 389-394.
6. Hosokawa, M., et al., Fucoxanthin regulates adipocytokine mRNA expression in white adipose tissue of diabetic/obese KK-Ay mice. Archives of biochemistry and biophysics, 2010. 504(1): p. 17-25.

Caffeine Anhydrous
1. Glaister, M., et al., Caffeine supplementation and multiple sprint running performance. Med Sci Sports Exerc, 2008. 40(10): p. 1835-40.
2. Schneiker, K.T., et al., Effects of caffeine on prolonged intermittent-sprint ability in team-sport athletes. Med Sci Sports Exerc, 2006. 38(3): p. 578-85.
3. Beaven, C.M., et al., Dose effect of caffeine on testosterone and cortisol responses to resistance exercise. Int J Sport Nutr Exerc Metab, 2008. 18(2): p. 131-41.
4. Astrup, A., et al., Caffeine: a double-blind, placebo-controlled study of its thermogenic, metabolic, and cardiovascular effects in healthy volunteers. The American journal of clinical nutrition, 1990. 51(5): p. 759-767.
5. Anderson, D.E. and M.S. Hickey, Effects of caffeine on the metabolic and catecholamine responses to exercise in 5 and 28 degrees C. Med Sci Sports Exerc, 1994. 26(4): p. 453-8.
6. Harpaz, E., et al., The effect of caffeine on energy balance. J Basic Clin Physiol Pharmacol, 2017. 28(1): p. 1-10.
7. Gurley, B.J., S.C. Steelman, and S.L. Thomas, Multi-ingredient, caffeine-containing dietary supplements: history, safety, and efficacy. Clin Ther, 2015. 37(2): p. 275-301.
8. Goldstein, E.R., et al., International society of sports nutrition position stand: caffeine and performance. J Int Soc Sports Nutr, 2010. 7(1): p. 5.
9. Spriet, L.L., Caffeine and performance. Int J Sport Nutr, 1995. 5 Suppl: p. S84-99.

N, N-Dimethylphenethylamine Citrate
1. Sotnikova, T.D., et al., Dopamine transporter-dependent and -independent actions of trace amine beta-phenylethylamine. J Neurochem, 2004. 91(2): p. 362-73.
2. Ishida, K., et al., Effects of beta-phenylethylamine on dopaminergic neurons of the ventral tegmental area in the rat: a combined electrophysiological and microdialysis study. J Pharmacol Exp Ther, 2005. 314(2): p. 916-22.
3. Kato, M., et al., β-Phenylethylamine modulates acetylcholine release in the rat striatum: involvement of a dopamine D 2 receptor mechanism. European journal of pharmacology, 2001. 418(1): p. 65-71.
4. Narang, D., et al., Trace amines and their relevance to psychiatry and neurology: a brief overview. Klinik Psikofarmakoloji Bülteni-Bulletin of Clinical Psychopharmacology, 2011. 21(1): p. 73-79.
5. Paterson, I., The potentiation of cortical neuron responses to noradrenaline by 2-phenylethylamine is independent of endogenous noradrenaline. Neurochemical research, 1993. 18(12): p. 1329-1336.
6. Shannon, H.E., E.J. Cone, and D. Yousefnejad, Physiologic effects and plasma kinetics of beta-phenylethylamine and its N-methyl homolog in the dog. J Pharmacol Exp Ther, 1982. 223(1): p. 190-6.
7. Ono, H., H. Ito, and H. Fukuda, 2-Phenylethylamine and methamphetamine enhance the spinal monosynaptic reflex by releasing noradrenaline from the terminals of descending fibers. The Japanese Journal of Pharmacology, 1991. 55(3): p. 359-366.

Teacrine (40% Tasteless Theacrine)
1. He, H., et al., Assessment of the Drug–Drug Interaction Potential Between Theacrine and Caffeine in Humans. Journal of caffeine research, 2017. 7(3): p. 95-102.
2. Dahlinghaus, M., Society of Sports Nutrition (ISSN) Conference and Expo. Journal of the International Society of Sports Nutrition, 2017. 14(2): p. P1.
3. Taylor, L., et al., Safety of TeaCrine®, a non-habituating, naturally-occurring purine alkaloid over eight weeks of continuous use. Journal of the International Society of Sports Nutrition, 2016. 13(1): p. 2.
4. Habowski, S., et al., The effects of TeacrineTM, a nature-identical purine alkaloid, on subjective measures of cognitive function, psychometric and hemodynamic indices in healthy humans: a randomized, double-blinded crossover pilot trial. Journal of the International Society of Sports Nutrition, 2014. 11(9008).
5. Ziegenfuss, T.N., et al., A two-part approach to examine the effects of theacrine (TeaCrine®) supplementation on oxygen consumption, hemodynamic responses, and subjective measures of cognitive and psychometric parameters. Journal of dietary supplements, 2017. 14(1): p. 9-24.
6. Hayward, S., et al., Safety of Teacrine®, a Non-Habituating, Naturally-Occurring Purine Alkaloid Over Eight Weeks of Continuous Use. Journal of the International Society of Sports Nutrition, 2015. 12(1): p. P59.

Hordeum Vulgare (std. to Hordenine)
1. Barwell, C.J., et al., Deamination of hordenine by monoamine oxidase and its action on vasa deferentia of the rat. J Pharm Pharmacol, 1989. 41(6): p. 421-3.
2. Nedergaard, O.A. and E. Westermann, Action of various sympathomimetic amines on the isolated stripped vas deferens of the guinea-pig. Br J Pharmacol, 1968. 34(3): p. 475-83.
3. Frank, M., et al., Hordenine: pharmacology, pharmacokinetics and behavioural effects in the horse. Equine Vet J, 1990. 22(6): p. 437-41.
4. Hapke, H.J. and W. Strathmann, [Pharmacological effects of hordenine]. Dtsch Tierarztl Wochenschr, 1995. 102(6): p. 228-32.
5. Pellati, F. and S. Benvenuti, Chromatographic and electrophoretic methods for the analysis of phenethylamine [corrected] alkaloids in Citrus aurantium. J Chromatogr A, 2007. 1161(1-2): p. 71-88.
6. Servillo, L., et al., Tyramine Pathways in Citrus Plant Defense: Glycoconjugates of Tyramine and Its N-Methylated Derivatives. J Agric Food Chem, 2017. 65(4): p. 892-899.
7. Konczol, A., et al., Blood-brain barrier specific permeability assay reveals N-methylated tyramine derivatives in standardised leaf extracts and herbal products of Ginkgo biloba. J Pharm Biomed Anal, 2016. 131: p. 167-174.

Huperzine-A (Huperzia Serrata 1% Extract)
1. Zhao, Q. and X.C. Tang, Effects of huperzine A on acetylcholinesterase isoforms in vitro: comparison with tacrine, donepezil, rivastigmine and physostigmine. European journal of pharmacology, 2002. 455(2): p. 101-107.
2. Kozikowski, A.P. and W. Tueckmantel, Chemistry, pharmacology, and clinical efficacy of the Chinese nootropic agent huperzine A. Accounts of chemical research, 1999. 32(8): p. 641-650.
3. Bai, D., Development of huperzine A and B for treatment of Alzheimer’s disease. Pure and applied chemistry, 2007. 79(4): p. 469-479.
4. Ved, H.S., et al., Huperzine A, a potential therapeutic agent for dementia, reduces neuronal cell death caused by glutamate. Neuroreport, 1997. 8(4): p. 963-967.
5. Peng, Y., et al., Huperzine A regulates amyloid precursor protein processing via protein kinase C and mitogen-activated protein kinase pathways in neuroblastoma SK-N-SH cells over-expressing wild type human amyloid precursor protein 695. Neuroscience, 2007. 150(2): p. 386-395.

Rauwolfia Vomitoria Extract (std. min. 90% rawolscine)
1. Perry, B.D. and D.C. U’Prichard, [3H]rauwolscine (alpha-yohimbine): a specific antagonist radioligand for brain alpha 2-adrenergic receptors. Eur J Pharmacol, 1981. 76(4): p. 461-4.
2. Rockhold, R.W. and F. Gross, Yohimbine diastereoisomers: cardiovascular effects after central and peripheral application in the rat. Naunyn Schmiedebergs Arch Pharmacol, 1981. 315(3): p. 227-31.
3. Arthur, J.M., S.J. Casanas, and J.R. Raymond, Partial agonist properties of rauwolscine and yohimbine for the inhibition of adenylyl cyclase by recombinant human 5-HT1A receptors. Biochem Pharmacol, 1993. 45(11): p. 2337-41.
4. Wainscott, D.B., et al., [3H]Rauwolscine: an antagonist radioligand for the cloned human 5-hydroxytryptamine2b (5-HT2B) receptor. Naunyn Schmiedebergs Arch Pharmacol, 1998. 357(1): p. 17-24.
5. Kohli, J.D. and N.N. De, Pharmacological action of rauwolscine. Nature, 1956. 177(4521): p. 1182.